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Registro Completo |
Biblioteca(s): |
Biblioteca Rui Tendinha. |
Data corrente: |
07/06/2021 |
Data da última atualização: |
07/06/2021 |
Tipo da produção científica: |
Folheto/Folha |
Autoria: |
ANGELETTI, M. da P. A.; OLIVEIRA, G. M. de; FIDELES, T. V.; MUNIZ, E. S.; COSTA, H.; MORAIS, F. M. de.; PILON, A. M.; MUZZI, E. de M.; LAURETT, L. |
Afiliação: |
Maria da Penha Angeletti, Incaper; Galderes Magalhães de Oliveira, Incaper; Talita Vieira Fideles, Incaper; Evelson Sanche Muniz, STRSMJ; Helcio Costa, Incaper; Fabio Morandi de Morais, Incaper; Anderson Martins Pilon, Incaper; Ernesto de Moraes Muzzi, STRSMJ; Lucineia Laurett, STRSMJ. |
Título: |
Que futuro você quer para sua terra? O sistema de plantio direto é um caminho seguro. |
Ano de publicação: |
2021 |
Fonte/Imprenta: |
Santa Maria de Jetibá, ES : Incaper / STRSMJ, maio de 2021. |
Idioma: |
Português |
Notas: |
Pôster temático. |
Conteúdo: |
Poster do projeto de plantio direto. |
Palavras-Chave: |
Sistema de Plantio Direto. |
Thesagro: |
Plantio Direto. |
Categoria do assunto: |
-- |
URL: |
https://biblioteca.incaper.es.gov.br/digital/bitstream/123456789/4202/1/poster-plantio-direto2.pdf
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Marc: |
LEADER 00776nam a2200241 a 4500 001 1023156 005 2021-06-07 008 2021 bl uuuu u0uu1 u #d 100 1 $aANGELETTI, M. da P. A. 245 $aQue futuro você quer para sua terra? O sistema de plantio direto é um caminho seguro.$h[electronic resource] 260 $aSanta Maria de Jetibá, ES : Incaper / STRSMJ, maio de 2021.$c2021 500 $aPôster temático. 520 $aPoster do projeto de plantio direto. 650 $aPlantio Direto 653 $aSistema de Plantio Direto 700 1 $aOLIVEIRA, G. M. de 700 1 $aFIDELES, T. V. 700 1 $aMUNIZ, E. S. 700 1 $aCOSTA, H. 700 1 $aMORAIS, F. M. de. 700 1 $aPILON, A. M. 700 1 $aMUZZI, E. de M. 700 1 $aLAURETT, L.
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Biblioteca Rui Tendinha (BRT) |
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Registro Completo |
Biblioteca(s): |
Biblioteca Rui Tendinha. |
Data corrente: |
17/04/2018 |
Data da última atualização: |
17/04/2018 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
A - 1 |
Autoria: |
MADRONERO, J.; RODRIGUES, S. P.; ANTUNES, T. F. S.; ABREU, P. M. V.; VENTURA, J. A.; FERNANDES, A. A. R.; FERNANDES, P. M. B. |
Afiliação: |
Johana Madroñero, UFES; Silas P. Rodrigues, UFES; Tathiana F. S. Antunes, UFES; Paolla M. V. Abreu, UFES; Jose Aires Ventura, Incaper; A. Alberto R. Fernandes, UFES; Patricia Machado Bueno Fernandes, UFES. |
Título: |
Transcriptome analysis provides insights into the delayed sticky disease symptoms in Carica papaya |
Ano de publicação: |
2018 |
Fonte/Imprenta: |
Plant Cell Reports, p. 1-14, 2018. |
Idioma: |
Português |
Conteúdo: |
Carica papaya plants develop the papaya sticky disease (PSD) as a result of the combined infection of papaya meleira virus (PMeV) and papaya meleira virus 2 (PMeV2), or PMeV complex. PSD symptoms appear only after C. papaya flowers. To understand the mechanisms involved in this phenomenon, the global gene expression patterns of PMeV complex-infected C. papaya at pre-and post-flowering stages were assessed by RNA-Seq. The result was 633 and 88 differentially expressed genes at pre- and post-flowering stages, respectively. At pre-flowering stage, genes related to stress and transport were up-regulated while metabolism-related genes were down-regulated. It was observed that induction of several salicylic acid (SA)-activated genes, including PR1, PR2, PR5, WRKY transcription factors, ROS and callose genes, suggesting SA signaling involvement in the delayed symptoms. In fact, pre-flowering C. papaya treated with exogenous SA showed a tendency to decrease the PMeV and PMeV2 loads when compared to control plants. However, pre-flowering C. papaya also accumulated transcripts encoding a NPR1-inhibitor (NPR1-I/NIM1-I) candidate, genes coding for UDP-glucosyltransferases (UGTs) and several genes involved with ethylene pathway, known to be negative regulators of SA signaling. At post-flowering, when PSD symptoms appeared, the down-regulation of PR-1 encoding gene and the induction of BSMT1 and JA metabolism-related genes were observed. Hence, SA signaling likely operates at the pre-flowering stage of PMeV complex-infected C. papaya inhibiting the development of PSD symptoms, but the induction of its negative regulators prevents the full-scale and long-lasting tolerance. MenosCarica papaya plants develop the papaya sticky disease (PSD) as a result of the combined infection of papaya meleira virus (PMeV) and papaya meleira virus 2 (PMeV2), or PMeV complex. PSD symptoms appear only after C. papaya flowers. To understand the mechanisms involved in this phenomenon, the global gene expression patterns of PMeV complex-infected C. papaya at pre-and post-flowering stages were assessed by RNA-Seq. The result was 633 and 88 differentially expressed genes at pre- and post-flowering stages, respectively. At pre-flowering stage, genes related to stress and transport were up-regulated while metabolism-related genes were down-regulated. It was observed that induction of several salicylic acid (SA)-activated genes, including PR1, PR2, PR5, WRKY transcription factors, ROS and callose genes, suggesting SA signaling involvement in the delayed symptoms. In fact, pre-flowering C. papaya treated with exogenous SA showed a tendency to decrease the PMeV and PMeV2 loads when compared to control plants. However, pre-flowering C. papaya also accumulated transcripts encoding a NPR1-inhibitor (NPR1-I/NIM1-I) candidate, genes coding for UDP-glucosyltransferases (UGTs) and several genes involved with ethylene pathway, known to be negative regulators of SA signaling. At post-flowering, when PSD symptoms appeared, the down-regulation of PR-1 encoding gene and the induction of BSMT1 and JA metabolism-related genes were observed. Hence, SA signaling likely operates at the pre-flow... Mostrar Tudo |
Thesaurus NAL: |
Carica papaya; Defense responses; Papaya meleira virus; Transcriptome Plant'virus interaction. |
Categoria do assunto: |
-- |
Marc: |
LEADER 02422naa a2200241 a 4500 001 1020018 005 2018-04-17 008 2018 bl uuuu u00u1 u #d 100 1 $aMADRONERO, J. 245 $aTranscriptome analysis provides insights into the delayed sticky disease symptoms in Carica papaya$h[electronic resource] 260 $c2018 520 $aCarica papaya plants develop the papaya sticky disease (PSD) as a result of the combined infection of papaya meleira virus (PMeV) and papaya meleira virus 2 (PMeV2), or PMeV complex. PSD symptoms appear only after C. papaya flowers. To understand the mechanisms involved in this phenomenon, the global gene expression patterns of PMeV complex-infected C. papaya at pre-and post-flowering stages were assessed by RNA-Seq. The result was 633 and 88 differentially expressed genes at pre- and post-flowering stages, respectively. At pre-flowering stage, genes related to stress and transport were up-regulated while metabolism-related genes were down-regulated. It was observed that induction of several salicylic acid (SA)-activated genes, including PR1, PR2, PR5, WRKY transcription factors, ROS and callose genes, suggesting SA signaling involvement in the delayed symptoms. In fact, pre-flowering C. papaya treated with exogenous SA showed a tendency to decrease the PMeV and PMeV2 loads when compared to control plants. However, pre-flowering C. papaya also accumulated transcripts encoding a NPR1-inhibitor (NPR1-I/NIM1-I) candidate, genes coding for UDP-glucosyltransferases (UGTs) and several genes involved with ethylene pathway, known to be negative regulators of SA signaling. At post-flowering, when PSD symptoms appeared, the down-regulation of PR-1 encoding gene and the induction of BSMT1 and JA metabolism-related genes were observed. Hence, SA signaling likely operates at the pre-flowering stage of PMeV complex-infected C. papaya inhibiting the development of PSD symptoms, but the induction of its negative regulators prevents the full-scale and long-lasting tolerance. 650 $aCarica papaya 650 $aDefense responses 650 $aPapaya meleira virus 650 $aTranscriptome Plant'virus interaction 700 1 $aRODRIGUES, S. P. 700 1 $aANTUNES, T. F. S. 700 1 $aABREU, P. M. V. 700 1 $aVENTURA, J. A. 700 1 $aFERNANDES, A. A. R. 700 1 $aFERNANDES, P. M. B. 773 $tPlant Cell Reports, p. 1-14, 2018.
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